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Diagnosis and management of childhood polycystic kidney disease
Thu, May 5, 2011
Uncategorized
Management of toxic ingestions with the use of renal replacement therapy
Thu, May 5, 2011
Uncategorized
Advances in our understanding of the pathogenesis of glomerular thrombotic microangiopathy
Thu, May 5, 2011
Uncategorized
A major cellular receptor in human Shiga toxin HUS is called
a. crry
b. Gb3
c. CD42
d. MCP
e. Factor I
The predominant pathway that is affected by atypical HUS is the
a. VEGF-A pathway
b. Lectin complement pathway
c. Alternative complement pathway
d. Classical complement pathway
e. Coagulation pathway
Which of the following are solid phase complement components?
a. Factor H
b. Factor I
c. MCP
d. C3
e. C4
After renal transplantation, in which of the following is there least likely to be a recurrence of HUS in the transplanted kidney?
a. Factor H mutations
b. Factor I mutations
c. C3 mutations
d. MCP mutations
e. Thrombomodulin mutations
The cause of pneumococcal-induced HUS is thought to be
a. Alterations in podocyte-derived VEGF-A
b. Abnormalities in the classical complement pathway
c. Thromboxane hyper-stimulation
d. Exposure of neuraminidase on the cell surfaces
e. Production of neuraminidase by the bacteria exposing the TF antigen
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Thu, May 5, 2011
Uncategorized